Positional differences in reactive hyperemia provide insight into initial phase of exercise hyperemia

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Positional differences in reactive hyperemia provide insight into initial phase of exercise hyperemia.

Studies have reported a greater blood flow response to muscle contractions when the limb is below the heart compared with above the heart, and these results have been interpreted as evidence for a skeletal muscle pump contribution to exercise hyperemia. If limb position affects the blood flow response to other vascular challenges such as reactive hyperemia, this interpretation may not be correc...

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Reactive hyperemia revisited.

To the Editor: We read with great interest Dr Huang’s study, published in Arteriosclerosis, Thrombosis, and Vascular Biology.1 We are in total agreement with the authors about the clinical relevance in the perioperative setting of the hyperemic flow velocity, measured by brachial artery Doppler ultrasound. Dr Huang suggests that reactive hyperemia increases the accuracy of the noninvasive vascu...

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Reactive hyperemia and cardiovascular risk.

Since the description of the classical cardiovascular risk factors by the Framingham group some 50 years ago,1 more recent efforts have explored the utility of biomarkers to further refine risk stratification. Although vascular imaging and biochemical markers have shown considerable promise,2 assessment of vascular function has particular appeal. Endothelial dysfunction is an attendant feature ...

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Reactive Hyperemia in Human Forearm Vessels

Background The role of nitric oxide (NO) in reactive hyperemia (RH) is not well known. We investigated whether NO plays a role in RH in human forearm vessels by examining the effects of N0-monomethyl-L-arginine (L-NMMA), a blocker of NO synthesis, on reactive hyperemic flow. Methods and Results Forearm blood flow (FBF) was measured by strain-gauge plethysmography with a venous occlusion techniq...

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Adenosine metabolism in canine myocardial reactive hyperemia.

In pentobrabital-anesthetized open chest dogs, myocardial adenosine content is elevated by 5 or 15 seconds of left coronary artery occlusion and falls exponentially to control levels during reactive hyperemia. The rate constants for adenosine dissipation are (mean +/- SEM): -0.08 +/- 0.01 and -0.034 +/- 0.007 sec-1 after 5- and 15-second occlusion, respectively. Kinetic analysis of the reactive...

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ژورنال

عنوان ژورنال: Journal of Applied Physiology

سال: 2015

ISSN: 8750-7587,1522-1601

DOI: 10.1152/japplphysiol.01253.2013